By Falk K.G.
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3 HEALTH EFFECTS Dermal Exposure No quantitative information was located regarding absorption of 1,2,3-trichloropropane in humans or animals following dermal exposure. 2). Since vapor exposure was unlikely due to occlusive covering of the treatment area, it can be concluded that dermal absorption occurs to some extent. 1 Inhalation Exposure No information was located regarding the distribution of 1,2,3-trichloropropane in humans or animals following inhalation exposure. 2 Oral Exposure No information was located regarding the distribution of 1,2,3-trichloropropane in humans following oral exposure.
The spleen and thyroid are less sensitive targets of intermediate-duration oral exposure in animals (NTP 1983a; Villeneuve et al. 1985), but the biological significance of effects observed in these organs is uncertain. Although intermediate-duration inhalation studies with animals are consistent with acute data in showing the respiratory system to be a target of 1,2,3-trichloropropane toxicity, intermediate-duration exposure levels that do not cause adverse respiratory (nasal) effects have not been determined.
1982; Volp et al. 1984). , mercapturic acids) can be anticipated, based on the metabolic pathways that have been identified for other halogenated alkanes. Chloroalkanes such as 1,2,3-trichloropropane undergo dehalogenation reactions catalyzed by cytochrome P-450 (Ivanetich et al. 1978; Salmon et al. 1981; Van Dyke et al. 1971). , radicals) can be formed from these reactions leading to protein and DNA adducts or lipid peroxidation. Conjugation with glutathione could result in formation of sulfur mustard-like compounds that are potential alkylating agents.